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Enzastaurin, MXD, and abemaciclib also notably increased nail germ lengths with average changes of approximately 306.36 63.54 m, 308.13 45.60 m, and 277.30 15.85 m, respectively, as illustrated in Fig

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These in vivo findings not only reinforce the causal role of the MAGI3--catenin axis in driving everolimus resistance but also provide compelling preclinical evidence that dual-pathway blockade is a viable and well-tolerated strategy to overcome this resistance